Kirandeep K. Deol

This study identifies vitamin B2 metabolism as a regulator of FSP1 stability, which prevents ferroptosis by maintaining FSP1 function through FAD cofactor binding.

This study identifies CLCC1 as a regulator of hepatic neutral lipid flux and nuclear pore complex assembly through CRISPR screens and functional analyses.

This paper demonstrates that ER membrane proteins can be degraded via cytosolic E3 ligase recruitment, bypassing ERAD and relying on VCP-mediated ubiquitin chain cleavage.