Author

Mona Foth

University of UtahORCID 0000-0001-6507-8114Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 2 Research Focus:drug response / drug discovery· 2 Approach:experimental assay· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Approach:genetic perturbation / CRISPR· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

Alison F. MunroiD· 1 shared Amanda JiangiD· 1 shared Andrew D. CampbelliD· 1 shared Aparna HegdeiD· 1 shared Brandon MurphyiD· 1 shared Bryan SerrelsiD· 1 shared Carly A. BeckeriD· 1 shared Catriona A. FordiD· 1 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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1 shared paperCancer Biology

Alison F. MunroORCID

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2 papers

Recent·Most cited

Cancer BiologyCancer ResearchPeer-reviewedMay 22, 2026

Genetic Drivers of Sensitivity or Resistance to RAS(ON) Multi-Selective Inhibitors in NRAS-Mutated Melanoma

Mona Foth, Wontak Kim, Kayla T. O’Toole…·Martin McMahon

This study identifies RAS(ON) inhibitors effective against NRAS-mutated melanoma and reveals resistance mechanisms through Ppia and Map2k1 mutations.

5 citationstop 1%

humanexperimental assay

𝕏 PostBluesky

Cancer BiologyCancer ResearchPeer-reviewedDec 5, 2024

A targetable PREX2/RAC1/PI3Kβ signalling axis confers resistance to clinically relevant therapeutic approaches in melanoma

Catriona A. Ford, Dana Koludrović, Patricia P. Centeno…·Andrew D. Campbell

This study identifies a druggable PREX2/RAC1/PI3Kβ signalling axis in BRAF-mutant melanoma that confers resistance to therapies and can be targeted to improve treatment outcomes.

8 citations

𝕏 PostBluesky