Joshua M. Kaplan

The paper identifies gem-4/Copine as a target of MEF-2 and CRH-1/CREB that reduces gap junction coupling and increases muscle excitability in C. elegans.

The paper shows that immature GABAergic motor neurons in C. elegans embryos transiently inhibit motion via both synaptic and non-synaptic GABA release before synapse formation.

This study identifies CASK and FARP as scaffolding proteins that localize post-synaptic ACh receptors, enhancing cholinergic transmission in C. elegans.

The paper describes the stereotyped behavioral maturation of C. elegans embryos, identifying a rhythmic quiescence phase driven by a specific neuron.

The paper shows that Go signaling in HSN neurons inhibits egg-laying activity and allows stretch-dependent modulation of egg laying in C. elegans.

This study investigates the tissue-specific functions of C. elegans JPH-1, showing its role in neurotransmission and muscle contraction, and its interaction with calcium channels.

This study shows that male pheromones in C. elegans enhance hermaphrodite cholinergic synaptic transmission at the neuromuscular junction, affecting locomotion and mating efficiency.

The paper shows that UNC-13L's C2A domain forms homodimers or heterodimers with RIM, and that heterodimerization regulates synaptic vesicle release probability but not priming.

This study shows that axonal mitochondria in C. elegans are necessary for neuropeptide secretion, and that oxidative phosphorylation, not calcium uptake, is required for this process.