Lingling Zhang

The paper shows that immune surveillance in germinal centers eliminates premalignant B-cells, but lymphoma-like mutations allow immune escape and progression to lymphoma.

This study identifies relapse-founding cancer persister cells in follicular lymphoma using a mouse model and shows their transcriptional program is conserved in humans, with potential therapeutic targets identified.

The paper investigates how repetition affects temporal integration in auditory cortex by analyzing single-unit activity in rats, finding that adaptation compresses preferred inter-click intervals while preserving population-level stability.

The paper shows that auditory sensitivity to sound changes is gated by temporal boundaries, with sensitivity peaking mid-sound and dropping near onset and offset.

This study identifies echo-facilitated temporal sensitivity (EFTS) as a mechanism where echoes enhance the auditory system's ability to detect rapid temporal changes in sound.

This study investigates predictive processing in macaque auditory-prefrontal circuits during an auditory oddball task, revealing hierarchical interactions between auditory and prefrontal cortices.

This study shows that walking in humans reduces sensitivity to fine auditory temporal changes but preserves some auditory responses, revealing how locomotion modulates auditory processing.

The paper models a macrophage-myofibroblast circuit to predict fibrosis types and shows targeting the myofibroblast autocrine loop reduces fibrosis after myocardial infarction.

This study investigates temporal integration in the human auditory system using EEG and finds that regular transitional click trains elicit distinct neural responses compared to irregular ones, with clinical implications for coma diagnosis.

The paper identifies millisecond-scale temporal configuration of sound as a distinct auditory feature, showing hierarchical encoding across the mammalian auditory pathway.

This study identifies mutations in the IgG heavy chain in follicular lymphoma and EZB DLBCL that enhance BCR signalling and oncogenicity by disrupting the IgG intracellular tail.