Author

Glaucia C. Furtado

Icahn School of Medicine at Mount SinaiORCID 0000-0001-8489-5697Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1 Organism:human· 1 Assay:proteomics· 1

Frequent Co-Authors

Zhengxiang HeiD· 2 shared Bo ShopsiniD· 1 shared Brittany M. MilleriD· 1 shared Erin E. ZwackiD· 1 shared Eugene Rudensky· 1 shared Huabao Xiong· 1 shared Isabel VargasiD· 1 shared Jeremiah J. FaithiD· 1 shared

Activity by Year

2018-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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2 shared papersImmunology

Zhengxiang HeORCID

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2 papers

Recent·Most cited

Heterozygosity for Crohn's Disease Risk Allele of ATG16L1 Protects against Bacterial Infection

Xiaomin Yao, Eugene Rudensky, Patricia K. Martin, Brittany M. Miller, Isabel Vargas, Erin E. Zwack, Keenan A. Lacey, Zhengxiang He, Gláucia C. Furtado, Sérgio A. Lira, Victor J. Torres, Bo Shopsin, Ken Cadwell

The paper shows that heterozygosity for the ATG16L1 T300A allele protects against Salmonella infection, while homozygosity increases susceptibility.

Immunity · Feb 25, 2024Peer-reviewedImmunology

Interleukin 22 mediates interleukin 23-induced pathology in newborn mice by disrupting the function of pancreatic and intestinal cells

Glaucia C. Furtado, Lili Chen, Valentina Strohmeier, Zhengxiang He, Madhura Deshpande, Scott K. Durum, Thomas M. Moran, Thomas Kraus, Huabao Xiong, Jeremiah J. Faith, Sergio A. Lira

This study shows that IL-23-induced pathology in newborn mice is mediated by IL-22, which disrupts pancreatic and intestinal cell function, leading to malabsorption and early death.

bioRxiv · Jul 2, 2018 · PreprintImmunology