Nikola Grujic

The paper identifies two separable neurochemical axes—orexin-driven arousal and dopamine-mediated utility—that govern task engagement in mice, with implications for drug effects and motivation disorders.

The paper identifies a neural circuit involving hypocretin/orexin neurons that rapidly suppresses hunger through a GABAergic relay, enabling dynamic regulation of hunger based on arousal.

The paper identifies orexin/hypocretin neurons as precise encoders of net body movement across behaviors and metabolic states, independent of head movement and nutritional state.

The paper investigates how hypothalamic hypocretin/orexin neurons regulate decision policy complexity through metabolic and neural signaling mechanisms in mice.

The paper identifies hypothalamic MCH cells as a neural module that tracks and controls exploratory rearing through interactions with locus coeruleus neurons.

This study investigates the role of hypothalamic orexin neurons in controlling pupil size, linking their activity to arousal and reward states through specific stimulation and in vivo recordings.

This study shows that orexin neurons decode blood glucose dynamics to drive adaptive behavior in mice by tracking glucose gradients and generating neural adaptations.

The paper investigates whether hypocretin/orexin neurons track sensory, cognitive, or motor information by analyzing their activity during sound cues and reward outcomes.

This study shows mice exhibit rational inattention by adaptively allocating sensory resources to maximize reward consumption while being imprecise for common stimuli.

This study investigates perceptual mislocalization during upward saccades and finds directional differences in peri-saccadic perceptual alterations.