Oren Kobiler

This study shows HSV-1 uses RAD51 to enhance viral gene expression by binding to promoter regions and interacting with ADNP.

This study shows that disrupting the brain-spleen axis accelerates disease progression in amyloidosis by impairing monocyte-microglia communication.

This study shows that antiviral drugs that partially inhibit HSV replication increase viral recombination and genetic rearrangements.

The paper identifies and characterizes monoclonal antibodies targeting the Mpox A28 protein, showing potent neutralization of Mpox and Vaccinia virus through complement-dependent mechanisms and providing protection in mice.

This study shows that herpesvirus recombination occurs when replication compartments coalesce, revealing spatial and temporal requirements for inter-genomic recombination.

The paper uses dynamic proteomics to study HSV1 infection in individual cells, finding preexisting cell-state differences influence infection outcomes.