Author

Marianthi Kiparaki

Albert Einstein College of MedicineORCID 0000-0002-0157-9064Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Developmental Biology· 2 Genetics· 1

Signature Assays, Organisms & Methods

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Research Focus:cell-cell communication· 3 Research Focus:differential expression· 3 Approach:experimental assay· 3 Research Focus:cell type annotation· 2 Organism:Drosophila· 2 Research Focus:gene regulatory network inference· 2 Approach:statistical model· 2 Organism:mouse· 1

Frequent Co-Authors

Jacky ChueniD· 2 shared Nicholas E. BakeriD· 2 shared Panagiotis MoulosiD· 2 shared Chaitali KhaniD· 1 shared Efthimios M. C. SkoulakisiD· 1 shared Eleni N. TsakiriiD· 1 shared Katerina DoukaiD· 1 shared Kiriaki Kanakousaki· 1 shared

Activity by Year

2021-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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2 shared papersDevelopmental Biology

Jacky ChuenORCID

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3 papers

Recent·Most cited

RpS12-mediated induction of the Xrp1short isoform links ribosomal protein mutations to cell competition

Eleni N. Tsakiri, Myrto Potiri, Kyriaki Kontogiannidi, Katerina Douka, Kiriaki Kanakousaki, Nikoleta Vavouraki, Maria Loizou, Panagiotis Moulos, Efthimios M. C. Skoulakis, Martina Samiotaki, Panagiota Kafasla, Marianthi Kiparaki

The paper shows that RpS12 induces the Xrp1short isoform in Rp+/- cells, which is necessary and sufficient for their elimination, challenging existing models of ribosomal protein-linked fitness surveillance.

Cell Reports · May 22, 2026Peer-reviewedDevelopmental Biology

The transcription factor Xrp1 orchestrates both reduced translation and cell competition upon defective ribosome assembly or function

Marianthi Kiparaki, Chaitali Khan, Virginia Folgado-Marco, Jacky Chuen, Panagiotis Moulos, Nicholas E. Baker

This study identifies Xrp1 as a master regulator that triggers cell competition and translation deficits in response to ribosomal stress.

eLife · Feb 18, 2022Peer-reviewedDevelopmental Biology

Cell competition removes segmental aneuploid cells from Drosophila imaginal disc-derived tissues based on ribosomal protein gene dose

Zhejun Ji, Jacky Chuen, Marianthi Kiparaki, Nicholas E. Baker

The paper shows that cell competition eliminates segmentally aneuploid cells in Drosophila imaginal discs through the RpS12-Xrp1 pathway, dependent on differences in ribosomal protein gene dose.

eLife · Apr 12, 2021Peer-reviewedGenetics