Author

Dawn H. W. Lau

Alzheimer’s Research UKORCID 0000-0002-0718-280XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:cell-cell communication· 1 Approach:experimental assay· 1 Organism:human· 1 Assay:proteomics· 1 Organism:rat· 1 Approach:statistical model· 1

Frequent Co-Authors

Beatriz Gomez Perez‐NievasiD· 1 shared Carmen M. NavarróniD· 1 shared Christina ElliottiD· 1 shared Claire TroakesiD· 1 shared Diane P. HangeriD· 1 shared Elizabeth B. C. GlennoniD· 1 shared Leela Phadke· 1 shared Lorenza MagnoiD· 1 shared

Activity by Year

2020-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperNeuroscience

Beatriz Gomez Perez‐NievasORCID

Mutual co-authors in cluster

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2 papers

Recent·Most cited

A primary rodent triculture model to investigate the role of glia-neuron crosstalk in regulation of neuronal activity.

Leela Phadke, Dawn H. W. Lau, Nozie D. Aghaizu, Shania Ibarra, Carmen M. Navarrón, Lucy Granat, Lorenza Magno, Paul John Whiting, Sarah Jolly

This paper develops a triculture model of neurons, astrocytes, and microglia to study glia-neuron interactions and their impact on neuronal activity and synapse function.

Frontiers in Aging Neuroscience · Dec 1, 2022Peer-reviewedNeuroscience

Loss of the Alzheimer’s-linked bridging integrator 1 (BIN1) protein affects synaptic structure and disrupts tau localisation and release

Elizabeth B. C. Glennon, Dawn H. W. Lau, Rebecca M. C. Gabriele, Matthew F Taylor, Claire Troakes, Sarah Opie-Martin, Christina Elliott, Richard Killick, Diane P. Hanger, Beatriz Gomez Perez‐Nievas, Wendy J. Noble

This study shows that BIN1 loss in Alzheimer's disease leads to tau mislocalization and altered synaptic structure and tau release.

Brain Communications · Jan 1, 2020Peer-reviewedNeuroscience