Author

Albano C. Méli

Centre National de la Recherche ScientifiqueORCID 0000-0002-1727-3027Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Biophysics· 1 Cell Biology· 1 Pathology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Organism:human· 4 Approach:experimental assay· 3 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Assay:proteomics· 2 Organism:rat· 2 Assay:bulk RNA-seq· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

Alain LacampagneiD· 2 shared Azzouz Charrabi· 2 shared Hugo Benoit· 2 shared Matteo Elia MangoniiD· 2 shared Pietro MesircaiD· 2 shared Alessandro PorroiD· 1 shared Ana Maria GomeziD· 1 shared Andrea SaponaroiD· 1 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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2 shared papersPhysiology

Alain LacampagneORCID

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4 papers

Recent·Most cited

PhysiologybioRxivPreprintSep 4, 2026

RyR2-Dependent Calcium Leak Drives the Acute Cardiovascular Manifestations of Ciguatera Poisoning: implications for an emerging climate-sensitive cardiotoxic disease

Hugo Benoit, Ismael Choukri, Pierre Sicard…·Albano C. Méli

This study identifies RyR2-dependent calcium leak as a mechanism underlying acute cardiovascular effects of ciguatoxin poisoning in human cardiomyocytes and rats.

DOI 10.64898/2026.08.31.748269

humanrat

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Cell BiologybioRxivPreprintJan 23, 2026

Validation of differentiated sinoatrial-like hiPSCs as a model of native sinus node myocytes

Eleonora Torre, Yvonne Sleiman, Haikel Dridi…·Albano C. Méli

The paper validates a protocol to generate sinoatrial-like hiPSC-derived cardiomyocytes that mimic native SAN myocytes in function and protein expression.

DOI 10.64898/2026.01.21.700785

humanproteomics

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BiophysicsbioRxivPreprintMar 20, 2025

Animal-free recombinant nanobody rescues HCN4 channel deficit in sinus node dysfunction

Atiyeh Sadat Sharifzadeh, Roberta Castelli, Alessandro Porro…·Anna Moroni

This study identifies a recombinant nanobody that specifically activates HCN4 channels and rescues sinus node dysfunction in a patient with a loss-of-function mutation.

DOI 10.1101/2025.03.20.644287

humanrat

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PathologybioRxivPreprintJan 10, 2025

The Mutated p.H222P A-type Lamins Drive Loxl2-Mediated Extracellular Matrix Remodeling in Both Patient-Derived Cardiomyocytes and Mouse Models of Dilated Cardiomyopathy

Marie Kervella, Cécile Peccate, Zoheir Guesmia…·Antoine Muchir

This study identifies Loxl2 as a therapeutic target for LMNA cardiomyopathy by showing its role in ECM remodeling in patient-derived cardiomyocytes and mouse models.

2 citations

humanmouse

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