Author

Brandon J. Vaglio

Rutgers, The State University of New JerseyORCID 0000-0002-2016-9212Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2 Genomics· 1

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 3 Approach:experimental assay· 3 Research Focus:gene regulatory network inference· 3 Organism:mouse· 3 Approach:statistical model· 3 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1

Frequent Co-Authors

Bonnie L. FiresteiniD· 3 shared Alana LoRe· 1 shared Alexia Tyler King· 1 shared Benjamin D. Buckley· 1 shared Brian P. DanielsiD· 1 shared Colm AtkinsiD· 1 shared Daniel H. GeschwindiD· 1 shared Dylan Sullivan· 1 shared

Activity by Year

2023-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 19 mutual co-author links

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3 shared papersNeuroscience

Bonnie L. FiresteinORCID

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3 papers

Recent·Most cited

A CK2α-G3BP1 signaling axis regulates local translation in developing neurons and is disrupted in OCNDS

Manasi Agrawal, Meghal Desai, Shruti Ghumra, Yashashree Bhorkar, Brandon J. Vaglio, Kyle Stokes, Krishna Rana, Ny-Ziah Hamilton Hill, Peace Nweze, Nethra Sriram, Alana LoRe, Riki Kawaguchi, Bonnie L. Firestein, Jack M. Parent, Daniel H. Geschwind, , Pabitra Kumar Sahoo

This study identifies a CK2α-G3BP1 signaling axis critical for neuronal translation and development, showing its disruption causes OCNDS pathology.

bioRxiv · Aug 19, 2026 · PreprintNeuroscience

RIPK3 promotes neuronal survival by suppressing excitatory neurotransmission during CNS viral infection

Irving Estevez, Benjamin D. Buckley, Nicholas Panzera, Marissa Lindman, Tsui-Wen Chou, Micheal McCourt, Brandon J. Vaglio, Colm Atkins, Bonnie L. Firestein, Brian P. Daniels

This study identifies RIPK3's role in promoting neuronal survival during CNS viral infection by suppressing excitatory neurotransmission, independent of its traditional necroptotic function.

bioRxiv · Apr 26, 2024 · PreprintNeuroscience

Cross-species transcriptomic analysis identifies mitochondrial dysregulation as a functional consequence of the schizophrenia-associated 3q29 deletion

Ryan H. Purcell, Esra Sefik, Erica Werner, Alexia Tyler King, Trenell J. Mosley, Megan E. Merritt-Garza, Pankaj Chopra, Zachary T. McEachin, Sridhar Karne, Nisha Raj, Brandon J. Vaglio, Dylan Sullivan, Bonnie L. Firestein, Kedamawit Tilahun, Maxine I. Robinette, , Jennifer Gladys Mulle

This study uses cross-species scRNA-seq to show that the 3q29 deletion causes mitochondrial dysfunction in both human cortical organoids and mouse models, suggesting a conserved metabolic disruption in schizophrenia.

Science Advances · Aug 16, 2023Peer-reviewedGenomics