Thomas P. Jensen

The paper shows that sensory experience increases glutamate release and strengthens synaptic connections in the barrel cortex, leading to changes in the functional connectome.

The paper improves a GABA sensor, iGABASnFR2, with higher sensitivity and faster kinetics, enabling better in vivo imaging of GABAergic transmission.

The paper shows that volume-transmitted GABA waves drive epileptiform rhythms in hippocampal networks by elevating tonic GABAA receptor conductance across multiple cells.

The paper shows that sensory-stimulation LTP increases synaptic fidelity while reducing excitation at thalamocortical connections, generating spatially intersecting glutamate discharges.

The paper shows that release probability increases with distance from the soma in CA3-CA1 synapses, enhancing signal transfer fidelity at higher frequencies.

This study shows that LTP induces astroglial withdrawal, increasing glutamate spillover and inter-synaptic communication.

This paper develops a genetically encoded fluorescent sensor, iGABASnFR, to monitor GABA in vivo and demonstrates its use in tracking GABA dynamics in various neural contexts.

The paper uses multiplex imaging to simultaneously measure presynaptic Ca2+ and glutamate release at multiple synapses in situ.

The paper constructs a realistic multi-compartmental astrocyte model to explore its biophysical properties and uncover features inaccessible through empirical methods.