Author

Jean‐François Fonteneau

Centre National de la Recherche ScientifiqueORCID 0000-0002-3284-8319Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:drug response / drug discovery· 2 Organism:mouse· 2 Assay:proteomics· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Approach:experimental assay· 1 Approach:genetic perturbation / CRISPR· 1 Organism:human· 1

Frequent Co-Authors

Joëlle S. NaderiD· 2 shared Nicolas BoisgeraultiD· 2 shared Aleksandr BarinoviD· 1 shared Anastassia V. KomarovaiD· 1 shared Atousa Arbabian· 1 shared Bernadette Brzezicha· 1 shared Camille Chatelain· 1 shared Chantal CombredetiD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 38 mutual co-author links

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2 shared papersImmunology

Joëlle S. NaderORCID

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2 papers

Recent·Most cited

A novel tumor-targeted interferon-α/-β receptor 1 antagonist increases replication of oncolytic vesicular stomatitis virus in a mouse mesothelioma model

Thomas Teja Ogor, Yann Bordat, Manon Souchard, Joëlle S. Nader, Geneviève Garcin, Camille Chatelain, Virginie Dehame, Sophie Deshayes, Lucas Treps, Mar Naranjo‐Gómez, Nicolas Boisgerault, Jan Tavernier, Mireia Pelegrín, Jean‐François Fonteneau

This paper develops IFNAR1 antagonists that selectively block IFNAR signaling in tumor cells, enhancing VSV oncolytic activity in mesothelioma and melanoma models.

bioRxiv · Jun 3, 2026 · PreprintImmunology

Disabling a key viral innate immunity antagonist converts measles vaccine into RIG-I-driven cancer immunotherapy

Aleksandr Barinov, Heidy Vera-Peralta, Joëlle S. Nader, Valérie Najburg, Chantal Combredet, Atousa Arbabian, Ségolène Gracias, Phanramphoei Namprachan Frantz, Roseline Vibert, Eddy Simard, Marie Coateval, Daniel Pouliquen, Tacien Petithomme, Matthieu Prot, Etienne Simon‐Lorière, , Frédéric Tangy

This paper modifies a measles vaccine by removing a viral protein to create a cancer immunotherapy that activates the immune system via RIG-I signaling, leading to tumor regression in multiple models.

bioRxiv · Oct 3, 2025 · PreprintImmunology