Author

Anne Mette G. Jensen

Aarhus UniversityORCID 0000-0002-3435-2691Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:differential expression· 2 Approach:experimental assay· 2 Research Focus:cell type annotation· 1 Organism:human· 1 Organism:mouse· 1 Organism:plant (other)· 1 Assay:proteomics· 1 Assay:scRNA-seq· 1

Frequent Co-Authors

Olav M. AnderseniD· 3 shared Charlotte Elisabeth TeunisseniD· 2 shared Giulia MontiiD· 2 shared A. Møller· 1 shared Aage Kristian Olsen AlstrupiD· 1 shared Aleksandra G. BilskaiD· 1 shared Anna R. MalikiD· 1 shared Anne Marlene LandauiD· 1 shared

Activity by Year

2022-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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3 shared papersNeuroscience

Olav M. AndersenORCID

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3 papers

Recent·Most cited

Soluble SORL1 in cerebrospinal fluid as a marker for functional impact of rare SORL1 variants.

Matthijs W. J. de Waal, Sven J. van der Lee, Melanie Lunding, Lynn Boonkamp, Nolan Barrett, Giulia Monti, Anne Mette G. Jensen, Christian Bjerggaard Vægter, Jan Raška, Sona Cesnarikova, Jiří Sedmík, Calvin Trieu, Marjan M. Weiss, Rosalina M.L. van Spaendonk, Lisa Vermunt, , Henne H. Holstege

The paper measures soluble SORL1 (sSORL1) in CSF to assess functional impact of rare SORL1 variants, finding lower sSORL1 in carriers of pathogenic variants.

Alzheimer s & Dementia · Feb 1, 2026Peer-reviewedNeuroscience

SorLA restricts TNFalpha release from microglia to shape glioma-supportive brain microenvironment

Paulina Kamińska, Peter Lund Ovesen, Mateusz Jakiel, Tomasz Obrębski, Vanessa Schmidt, Michał Dramiński, Aleksandra G. Bilska, Magdalena Bieniek, Jasper J. Anink, Bohdan Paterczyk, Anne Mette G. Jensen, Sylwia Piątek, Olav M. Andersen, Eleonora Aronica, Thomas E. Willnow, , Anna R. Malik

The paper shows SorLA restricts TNFα release from microglia, reducing their anti-tumor response and promoting glioma growth.

EMBO Reports · Mar 18, 2024Peer-reviewedNeuroscience

In vivo evidence that SORL1, encoding the endosomal recycling receptor SORLA, can function as a causal gene in Alzheimer's Disease

Olav M. Andersen, Nikolaj Bøgh, Anne Marlene Landau, Gro Grunnet Pløen, Anne Mette G. Jensen, Giulia Monti, Benedicte Parm Ulhøi, Jens Randel Nyengaard, Kirsten Rosenmay Jacobsen, Arne Lund Jørgensen, Ida E. Holm, Marianne Kristensen, Aage Kristian Olsen Alstrup, Esben Søvsø Szocska Hansen, Charlotte Elisabeth Teunissen, , Charlotte Brandt Sørensen

This study shows that SORL1 haploinsufficiency in pigs mimics early AD biomarker changes without neurodegeneration, supporting its role as a causal gene in Alzheimer's disease.

Cell Reports Medicine · Sep 1, 2022Peer-reviewedNeuroscience