Author

Kunal Shroff

University of California, San FranciscoORCID 0000-0002-3803-7517Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 2 Research Focus:cell type annotation· 1 Assay:CRISPR screen· 1 Research Focus:differential expression· 1 Organism:human· 1 Research Focus:perturbation response prediction· 1 Assay:proteomics· 1

Frequent Co-Authors

Martin KampmanniD· 2 shared Amber L. Cramer· 1 shared Céline EidenschenkiD· 1 shared Chao CheniD· 1 shared Claire G. JeongiD· 1 shared Emily MarzetteiD· 1 shared Gregory A. MohliD· 1 shared Indigo V.L. RoseiD· 1 shared

Activity by Year

2021-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersNeuroscience

Martin KampmannORCID

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3 papers

Recent·Most cited

NeurosciencebioRxivPreprintJun 30, 2026

Synaptic activity controls local exposure of an 'eat-me' signal via ANO3-ITPR1 signaling

Kunal Shroff, Molly C O'Brien, Tyler J. Smith…·Martin Kampmann

The paper identifies ANO3-ITPR1 signaling as a caspase-independent mechanism for synaptic phosphatidylserine exposure, which controls synapse pruning during development and neurodegeneration.

1 citation

mouseexperimental assay

𝕏 PostBluesky

NeurosciencebioRxivPreprintFeb 28, 2024

A Massively Parallel CRISPR-Based Screening Platform for Modifiers of Neuronal Activity

Steven C. Boggess, Vaidehi Gandhi, Ming‐Chi Tsai…·Martin Kampmann

The paper develops a CRISPRi-based screening platform to identify genes affecting neuronal excitability in human iPSC-derived neurons, revealing new links to neurodegenerative and neurodevelopmental disorders.

6 citations

humanCRISPR screen

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NeuroscienceNeurobiology of DiseasePeer-reviewedAug 3, 2021

DYT-TOR1A Subcellular Proteomics Reveals Selective Vulnerability of the Nuclear Proteome to Cell Stress

Kunal Shroff, Zachary F. Caffall, Nicole Calakos

This study uses subcellular proteomics to show that DYT-TOR1A mutant cells have greater nuclear proteome disruption under stress compared to other conditions.

17 citations

mouseproteomics

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