Author

Emily Marzette

University of California, San FranciscoORCID 0000-0002-1010-5848Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 3 Research Focus:differential expression· 2 Assay:ATAC-seq· 1 Assay:bulk RNA-seq· 1 Research Focus:cell-cell communication· 1 Assay:CRISPR screen· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

Martin KampmanniD· 3 shared Andrea Keys· 2 shared Andrew Li· 2 shared Danielle L. SwaneyiD· 2 shared Gary DixoniD· 2 shared Gregory A. MohliD· 2 shared Julianne JiniD· 2 shared Justin McKetneyiD· 2 shared

Activity by Year

2024-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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3 shared papersNeuroscience

Martin KampmannORCID

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3 papers

Recent·Most cited

Tau is an m6A-associated RNA binding protein that causes RNA dysregulation in disease

Gary Dixon, Andrea Keys, Katherine L. Rothamel, Justin McKetney, Yi Xiao Jiang, Lacey A. Gordon, Madeline L. Page, Chang Liu, Katrina Hou, Keegan Flanagan, Brian Yee, Evan Xie, Gregory A. Mohl, Emily Marzette, Julianne Jin, , Martin Kampmann

The paper identifies tau as an m6A-associated RNA binding protein that causes RNA dysregulation in MAPT coding-variant patients and V337M iNeurons.

bioRxiv · Oct 29, 2026 · PreprintMolecular Biology

Multi-omic phenotyping of MAPT V337M neurons reveals early changes in axonogenesis and tau phosphorylation

Gregory A. Mohl, Gary Dixon, Emily Marzette, Justin McKetney, Avi J. Samelson, Carlota Pereda Serras, Julianne Jin, Nabeela Ariqat, Andrea Keys, Cristian Chavira, Andrew Li, Steven C. Boggess, Danielle L. Swaney, Martin Kampmann

The paper uses multi-omic analysis to study MAPT V337M neurons, finding altered axonogenesis and tau phosphorylation, with p38 MAPK pathway involvement in tau phosphorylation in V337M neurons.

npj Dementia · Apr 25, 2026Peer-reviewedNeuroscience

A Massively Parallel CRISPR-Based Screening Platform for Modifiers of Neuronal Activity

Steven C. Boggess, Vaidehi Gandhi, Ming‐Chi Tsai, Emily Marzette, Noam Teyssier, Joanna Yu-Ying Chou, Xiaoyu Hu, Amber L. Cramer, Lin Yadanar, Kunal Shroff, Claire G. Jeong, Céline Eidenschenk, Jesse E. Hanson, Ruilin Tian, Martin Kampmann

The paper develops a CRISPRi-based screening platform to identify genes affecting neuronal excitability in human iPSC-derived neurons, revealing new links to neurodegenerative and neurodevelopmental disorders.

bioRxiv · Feb 28, 2024 · PreprintNeuroscience