Andreas Ritzau‐Jost

The paper shows that chronic inactivity does not broadly affect action potentials in neocortical neurons but does in hippocampal neurons under specific conditions.

This study investigates how LGI1 autoantibodies enhance synaptic transmission by reducing Kv1 channel density and broadening presynaptic action potentials.

This study shows that LTP at layer 5 pyramidal neurons increases synaptic strength by increasing the number of fully-primed, slowly-recovering vesicles.

This study develops NMDA-receptor-Fc-fusion constructs that neutralize anti-NMDA receptor antibodies and inhibit their effects on neurons.

This study shows that neuronal activity drives the translocation of LGI1 and ADAM23 to the synaptic cleft, adjusting excitatory neurotransmission based on activity history.

The paper reports direct recordings of presynaptic action potentials in neocortical boutons, showing differential broadening in excitatory and inhibitory neurons.