Author

Sarah L. Anzick

National Institutes of HealthORCID 0000-0002-4907-513XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 3 Organism:mouse· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Organism:non-human primate· 1 Assay:spatial transcriptomics· 1

Frequent Co-Authors

Craig MartensiD· 3 shared Aaron CarmodyiD· 1 shared Alea C. DelmastroiD· 1 shared Alex KongiD· 1 shared Alyssa B. EvansiD· 1 shared Amanda Griffin· 1 shared Andrea MarziiD· 1 shared Arthur WickenhageniD· 1 shared

Activity by Year

2025-2025 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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3 shared papersImmunology

Craig MartensORCID

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3 papers

Recent·Most cited

Orthobunyavirus neurovirulence is a complex trait involving all three genome segments

Matthew J. Abbott, Christina R. Norsten, Sarah L. Anzick, Craig Martens, Clayton W. Winkler, Alyssa B. Evans

This study identifies that LACV neurovirulence is a complex trait mediated by proteins on all three genome segments, with the M and S segments being primary drivers.

bioRxiv · Sep 16, 2025 · PreprintMicrobiology

Jamaican fruit bats (Artibeus jamaicensis) competence for Ebola virus but not Marburg virus is driven by intrinsic differences in viral entry and IFN-I signaling antagonism.

Sarah van Tol, Julia Rebecca Port, Robert J. Fischer, Shane Gallogly, Trenton Bushmaker, Amanda Griffin, Jonathan E. Schulz, Aaron Carmody, Lara M. Myers, Daniel E. Crowley, Caylee Falvo, Jade C. Riopelle, Arthur Wickenhagen, Chad S. Clancy, Jamie Lovaglio, , Vincent Jacobus Munster

This study shows Jamaican fruit bats can support EBOV replication and shedding but not MARV, due to differences in viral entry and IFN-I signaling suppression.

Nature Communications · Mar 25, 2025Peer-reviewedImmunology

The immunometabolic topography of tuberculosis granulomas governs cellular organization and bacterial control

Erin McCaffrey, Alea C. Delmastro, Isobel Fitzhugh, Jolene S. Ranek, Sarah Douglas, Joshua M. Peters, Christine Camacho Fullaway, Marc MB Bosse, Candace C. Liu, Craig Gillen, Noah F. Greenwald, Sarah L. Anzick, Craig Martens, Seth Winfree, Yunhao Bai, , Michael Angelo

This study identifies hypoxia as a key driver of immune dysfunction in TB granulomas, correlating with bacterial persistence.

bioRxiv · Feb 18, 2025 · PreprintImmunology