Author

Wei-Che Tseng

Emory UniversityORCID 0000-0002-5280-8074Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cell Biology· 1 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Research Focus:chromatin & epigenetic regulation· 1 Assay:proteomics· 1

Frequent Co-Authors

Kyle M. MilleriD· 2 shared Allyson E. Koyen· 1 shared Anna Marie KenneyiD· 1 shared B WuiD· 1 shared Baylee A. SmithiD· 1 shared Bing YaoiD· 1 shared Blerta XhemalçeiD· 1 shared Boya Gao· 1 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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2 shared papersMolecular Biology

Kyle M. MillerORCID

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2 papers

Recent·Most cited

HELZ is a RNA-DNA helicase that resolves R loops to facilitate homologous recombination repair

Ramona Haji‐Seyed‐Javadi, Allyson E. Koyen, Sandip Kumar Rath, B Wu, Matthew Z. Madden, Yingzi Hou, Priya Kapoor-Vazirani, Roshika Roshika, Meili Aiello, Nho Cong Luong, Wei-Che Tseng, Fatmata Sesay, J G Kim, Tony Tan, Seohyun Kim, , David S. Yu

This study identifies HELZ as an RNA-DNA helicase that resolves R loops to support homologous recombination repair and genomic stability.

Nature Communications · Jul 23, 2026Peer-reviewedMolecular Biology

TRIM24 directs replicative stress responses to maintain ALT telomeres via chromatin signaling

Daein Kim, Ragini Bhargava, Shih-Chun Wang, Wei-Che Tseng, Doohyung Lee, Riya Patel, Sungtaek Oh, Ray W. Bowman, Baylee A. Smith, Min Kyu Kim, Chan Hyun Na, Roderick J. O’Sullivan, Kyle M. Miller

This study identifies TRIM24's role in ALT telomere maintenance through chromatin signaling, showing it promotes telomere DNA synthesis via SUMO-dependent mechanisms.

Molecular Cell · Jan 1, 2024Peer-reviewedCell Biology