Diego Rayes

This study shows that microexon alternative splicing in C. elegans changes with developmental stage and is influenced by food availability and DNA topology, affecting feeding behavior.

This study uses a C. elegans model to show that cytoplasmic TDP-43 causes early functional impairments in serotonergic neurons without neurodegeneration.

This study shows that β-hydroxybutyrate improves GABAergic neurodevelopment in daf-18/PTEN mutants by enhancing DAF-16/FOXO activity.

This study identifies INS-3 as a key insulin-like peptide modulated by tyramine that influences stress resistance and reproductive development in C. elegans.

This study identifies a gain-of-function mutation in the UNC-2/CaV2α channel in C. elegans that causes hyperactivity and excitation-dominant synaptic transmission by altering cholinergic and GABAergic transmission.

The paper shows that a neurotransmitter receptor in C. elegans is co-opted for inter-organismal communication via pheromone sensing.

The paper shows that acute stress impairs cytoprotective mechanisms by inhibiting the insulin pathway through neural release of tyramine in C. elegans.