Author

Mayfong Mayxay

Ministry of HealthORCID 0000-0002-6056-4516Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Evolutionary Biology· 1 Systems Biology· 1

Signature Assays, Organisms & Methods

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Organism:plant (other)· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Assay:DNA methylation· 1 Approach:genetic perturbation / CRISPR· 1 Research Focus:phylogenetics / evolution· 1 Research Focus:variant calling· 1

Frequent Co-Authors

Abul Faiz· 2 shared Aung Pyae PhyoiD· 2 shared Chanaki Amaratunga· 2 shared Mallika ImwongiD· 2 shared Olivo MiottoiD· 2 shared Sasithon PukrittayakameeiD· 2 shared Tran Tinh HieniD· 2 shared Abdoulaye Djimdé· 1 shared

Activity by Year

2015-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersSystems Biology

Abul Faiz

Mutual co-authors in cluster

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2 papers

Recent·Most cited

The mechanism of artemisinin resistance of Plasmodium falciparum malaria parasites originates in their initial transcriptional response.

Lei Zhu, Rob W. van der Pluijm, Michal Kucharski, Sourav Nayak, Jaishree Tripathi, François Henry Nosten, Abul Faiz, Chanaki Amaratunga, Dysoley Lek, Elizabeth A. Ashley, Frank M. Smithuis, Aung Pyae Phyo, Khin Lin, Mallika Imwong, Mayfong Mayxay, , Zbynek Bozdech

The paper identifies a transcriptional profile in artemisinin-resistant Plasmodium falciparum that originates from initial drug responses and involves proteotoxic stress and REDOX metabolism.

bioRxiv · May 17, 2021 · PreprintSystems Biology

Genomic epidemiology of the current wave of artemisinin resistant malaria

Roberto Amato, Olivo Miotto, Charles Woodrow, Jacob Almagro-Garcia, Ipsita Sinha, Susana Campino, Daniel Mead, Eleanor Drury, Mihir Kekre, Mandy Sanders, Alfred Amambua-Ngwa, Chanaki Amaratunga, Lucas Amenga-Etego, Tim J C Anderson, Voahangy Andrianaranjaka, , MalariaGEN Plasmodium falciparum Community Project

This study identifies kelch13 mutations associated with artemisinin resistance in Southeast Asia and finds low frequency of these mutations in Africa, suggesting different evolutionary pressures.

bioRxiv · May 24, 2015 · PreprintEvolutionary Biology