Grant D. Schauer

This study shows Mrc1 balances replication fork stability and flexibility by linking CMG helicase and Pol2, and its phosphorylation alters this balance under checkpoint activation.

This paper shows DNA Polymerase α can replicate through bulky DNA lesions in vitro, but its TLS activity is suppressed by replisome components during normal replication.

This paper revises the mechanism of hydroxyurea-induced cell cycle arrest in yeast, showing it is ROS-mediated rather than Mrc1-dependent, and introduces an improved alternative to HU called RNR-deg.