Author

Alex Griffiths

King's College LondonORCID 0000-0002-6772-2054Unclaimed ORCID profile · follow to get notified of new preprints

Additional preprints under this name without ORCID

Alex Griffiths1 paper · Cardiff University

Biology Disciplines

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Neuroscience· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Assay:imaging· 1 Assay:metabolomics· 1 Assay:proteomics· 1

Frequent Co-Authors

Alexander P. MorrelliD· 1 shared Alexis PenverneiD· 1 shared Aubrey ConverseiD· 1 shared Benjamin J. O’CallaghaniD· 1 shared Christer HögstrandiD· 1 shared Darija ŠoltićiD· 1 shared Hannah J Lucas-ClarkeiD· 1 shared Hélène Plun‐FavreauiD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperPhysiology

Alexander P. MorrellORCID

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2 papers

Recent·Most cited

Deficiency of the membrane androgen receptor ZIP9 alters brain zinc distribution, reproductive endocrinology, and female fertility

Rui Wang, Rhiannon E. Boseley, Kalotina Geraki, Alexander P. Morrell, Alex Griffiths, Aubrey Converse, Peter Thomas, Kim Jonas, Robert Hindges, Christer Högstrand

This study shows that ZIP9 deficiency in zebrafish disrupts zinc distribution, reproductive endocrinology, and female fertility.

bioRxiv · May 8, 2026 · PreprintPhysiology

The SNCA A53T mutation sensitizes human neurons and microglia to ferroptosis

Laura Mahoney‐Sanchez, Hannah J Lucas-Clarke, Alexis Penverne, James R. Evans, Karishma D’Sa, Stephanie Strohbuecker, Patricia Lopez-Garcia, Katharina Cosker, Darija Šoltić, Benjamin J. O’Callaghan, Alex Griffiths, Sean A. Pintchovski, Hélène Plun‐Favreau, Jenny Hällqvist, Kevin A. Mills, Sonia Gandhi

This study shows that the SNCA A53T mutation increases vulnerability to ferroptosis in neurons and microglia, suggesting 15-LO inhibition as a potential therapeutic target for Parkinson's disease.

bioRxiv · Oct 13, 2025 · PreprintNeuroscience