Author

Stephen P. Messier

Wake Forest UniversityORCID 0000-0002-7033-8459Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Organism:human· 1 Assay:metabolomics· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

Antonina M. AkkiD· 1 shared Arin K. OestreichiD· 1 shared Ashley N. PlumbiD· 1 shared Bethany A. AndokoiD· 1 shared Bo ZhangiD· 1 shared Christine T. N. PhamiD· 1 shared Darren DumlaoiD· 1 shared Farshid GuilakiD· 1 shared

Activity by Year

2026-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperPhysiology

Antonina M. AkkORCID

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1 paper

Recent·Most cited

Adipose-driven complement-lipid reprogramming controls nociceptive vulnerability in obesity-associated osteoarthritis

Hope D. Welhaven, Joseph B. Lesnak, Kristin L. Lenz, Bethany A. Andoko, Arin K. Oestreich, Ashley N. Plumb, Katelyn E. Sadler, Luke E. Springer, Jacob G. Brockert, Darren Dumlao, Antonina M. Akk, Xiaobo Wu, Huimin Yan, Bo Zhang, Stephen P. Messier, , Kelsey H. Collins

The paper identifies an adipose-complement-lipid axis that systemically regulates sensory neuron sensitization, explaining pain-structure discordance in obesity-associated osteoarthritis.

bioRxiv · Mar 21, 2026 · PreprintPhysiology