Author

Tatiana Brémovà-Ertl

University Hospital of BernORCID 0000-0002-7397-045XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Organism:mouse· 2 Research Focus:benchmarking· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

Michael Leo StruppiD· 2 shared Alessio Vittorio ColomboiD· 1 shared Andrew P. LiebermaniD· 1 shared Barry BolandiD· 1 shared Claire SmithiD· 1 shared David Aitchison SmithiD· 1 shared Ecem KayaiD· 1 shared Frances M. PlattiD· 1 shared

Activity by Year

2020-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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2 shared papersNeuroscience

Michael Leo StruppORCID

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2 papers

Recent·Most cited

Loss of NPC1 enhances phagocytic uptake and impairs lipid trafficking in microglia

Alessio Vittorio Colombo, Lina Dinkel, Stephan A. Müller, Laura Sebastián Monasor, Martina Schifferer, Ludovico Cantuti‐Castelvetri, Jasmin König, Lea Vidatic, Tatiana Brémovà-Ertl, Andrew P. Lieberman, Silva Katusic Hecimovic, Mikael Simons, Stefan F. Lichtenthaler, Michael Leo Strupp, Susanne A. Schneider, Sabina Tahirović

This study shows that loss of NPC1 in microglia increases phagocytic uptake and impairs lipid trafficking, leading to myelin accumulation and potential therapeutic implications.

Nature Communications · Feb 24, 2021Peer-reviewedNeuroscience

Acetyl-Leucine slows disease progression in lysosomal storage disorders

Ecem Kaya, David Aitchison Smith, Claire Smith, Lauren Morris, Tatiana Brémovà-Ertl, Mario Cortina‐Borja, Paul Fineran, Karl Morten, Joanna Poulton, Barry Boland, John Spencer, Michael Leo Strupp, Frances M. Platt

This study shows that ADLL and its enantiomer ALL slow disease progression in lysosomal storage disorders through mechanisms involving glucose and antioxidant metabolism.

Brain Communications · Sep 15, 2020Peer-reviewedNeuroscience