Author

Alice Nevone

University of PaviaORCID 0000-0002-7724-1043Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1 Assay:proteomics· 1

Frequent Co-Authors

Mario NuvoloneiD· 2 shared Anthony FrosioiD· 1 shared Carlo PapponeiD· 1 shared Caroline DemangeliD· 1 shared Chloé Veront· 1 shared Daniela RicciiD· 1 shared Dario MelgariiD· 1 shared David HardyiD· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersCell Biology

Mario NuvoloneORCID

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2 papers

Recent·Most cited

Targeting Sec61 to Silence Pathogenic Light-Chain Production in AL Amyloidosis

Daniela Ricci, Chloé Veront, Irene VALCOZZENA, Merlin Després, Alice Nevone, Marine Oberkampf, Mariette Matondo, Karen Druart, Thibaut Douché, Sarra Loulizi, David Hardy, Florence Bugault, Manon Capdeville, Mario Nuvolone, Caroline Demangel

This study targets the Sec61 translocon to suppress pathogenic light-chain production in AL amyloidosis, showing that mycolactone and synthetic inhibitors like A317 reduce LC secretion while highlighting the need for client selectivity.

bioRxiv · Sep 13, 2026 · PreprintCell Biology

Exploring light chain cardiotoxicity in AL amyloidosis: Impact on hiPSC-derived Cardiomyocyte Activity

Serena Calamaio, Anthony Frosio, Dario Melgari, Luca Broggini, Federica Sonzini, Rachele Prevostini, Davide Pignatelli, Luigi Anastasia, Carlo Pappone, Alice Nevone, Giovanni O. Palladini, Mario Nuvolone, Stéfano Ricagno, Ilaria Rivolta

This study uses hiPSC-derived cardiomyocytes to show that cardiotoxic light chains reduce electrophysiological function, while non-toxic light chains have no effect.

Amyloid · Jun 19, 2026Peer-reviewedPathology