Author

Cristiano Petrini

IFOMORCID 0000-0002-7727-7230Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Assay:ChIP-seq· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Research Focus:gene regulatory network inference· 2 Organism:human· 2 Research Focus:spatial domain detection· 2 Assay:ATAC-seq· 1 Assay:bulk RNA-seq· 1

Frequent Co-Authors

Chiara LanzuoloiD· 2 shared Endre SebestyéniD· 2 shared Federica LuciniiD· 2 shared Francesco FerrariiD· 2 shared Adnan AlaziziiD· 1 shared Alexander G Shanku· 1 shared Allison L. RichardsiD· 1 shared Andrea BianchiiD· 1 shared

Activity by Year

2019-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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2 shared papersGenomics

Chiara LanzuoloORCID

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3 papers

Recent·Most cited

Biochemical properties of chromatin domains define genome compartmentalization

Federica Lucini, Cristiano Petrini, Elisa Salviato, Koustav Pal, Valentina Rosti, Francesca Gorini, Philina Santarelli, Roberto Quadri, Giovanni Lembo, Giulia Graziano, Emanuele Di Patrizio Soldateschi, Ilario Tagliaferri, Eva Pinatel, Endre Sebestyén, Luca Rotta, , Francesco Ferrari

The paper develops 4f-SAMMY-seq to map euchromatin and heterochromatin based on accessibility and solubility, and reconstructs their 3D compartmentalization in one experiment.

Nucleic Acids Research · May 16, 2024Peer-reviewedGenomics

Early Polycomb-target deregulations in Hutchinson-Gilford Progeria Syndrome revealed by heterochromatin analysis

Endre Sebestyén, Fabrizia Marullo, Federica Lucini, Cristiano Petrini, Andrea Bianchi, Sara Valsoni, Ilaria Olivieri, Laura Antonelli, Francesco Gregoretti, Gennaro Oliva, Francesco Ferrari, Chiara Lanzuolo

The paper identifies early chromatin structural changes in HGPS using a new method, showing Polycomb target gene deregulation linked to lamin-associated domain disruption.

Nature Communications · Dec 8, 2020Peer-reviewedMolecular Biology

Interpreting coronary artery disease risk through gene-environment interactions in gene regulation

Anthony S Findley, Allison L. Richards, Cristiano Petrini, Adnan Alazizi, Elizabeth R. Doman, Alexander G Shanku, Gordon O. Davis, N. Hauff, Yoram Sorokin, Xiaoquan Wen, Roger Piqué-Regi, Francesca Luca

The paper investigates gene-environment interactions in coronary artery disease by analyzing RNA-seq and ATAC-seq data from endothelial cells exposed to environmental factors, finding links between environmental responses and CAD risk.

Genetics · Sep 6, 2019Peer-reviewedGenomics