Author

Elisabeth Baumann

Max Delbrück CenterORCID 0000-0002-7804-4885Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Organism:mouse· 2 Approach:statistical model· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:trajectory inference· 1

Frequent Co-Authors

Holger GerhardtiD· 2 shared Olya OppenheimiD· 2 shared Wolfgang GieseiD· 2 shared Andranik IvanoviD· 1 shared André RosaiD· 1 shared Anne EichmanniD· 1 shared Dieter BeuleiD· 1 shared Eireen Bartels‐Klein· 1 shared

Activity by Year

2023-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersDevelopmental Biology

Holger GerhardtORCID

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2 papers

Recent·Most cited

Gene-specific endothelial programs drive AVM pathogenesis in SMAD4 and ALK1 loss-of-function

Olya Oppenheim, Wolfgang Giese, Hyojin Park, Elisabeth Baumann, Andranik Ivanov, Dieter Beule, Anne Eichmann, Holger Gerhardt

This study identifies gene-specific mechanisms of AVM formation in endothelial cells with SMAD4 or ALK1 loss-of-function, showing distinct molecular and cellular pathways leading to different vascular malformations.

Arteriosclerosis Thrombosis and Vascular Biology · Jul 16, 2026Peer-reviewedDevelopmental Biology

Reconstructing stochastic cell population trajectories reveals regulators and heterogeneity of endothelial flow-migration coupling driving vascular remodelling

Wolfgang Giese, André Rosa, Elisabeth Baumann, Olya Oppenheim, Emir Bora Akmeriç, Santiago Andrade, Irene Hollfinger, Eireen Bartels‐Klein, Silvanus Alt, Holger Gerhardt

The paper develops a computational model to analyze EC migration during vascular remodelling, finding that directional migration is driven by dual force fields and mediated by Cdc42 with cell population heterogeneity.

bioRxiv · May 30, 2023 · PreprintDevelopmental Biology