Author

Justin Vercellino

Albert Einstein College of MedicineORCID 0000-0002-8286-4376Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Organism:human· 1 Assay:proteomics· 1

Frequent Co-Authors

Beata MałachowskaiD· 1 shared Brett I. BelliD· 1 shared Chandan GuhaiD· 1 shared David ShechteriD· 1 shared Dejauwne L. YoungiD· 1 shared Jacob S. RothiD· 1 shared Jennifer T. AguilaniD· 1 shared Joseph D. DeAngeloiD· 1 shared

Activity by Year

2025-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperCancer Biology

Beata MałachowskaORCID

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1 paper

Recent·Most cited

Loss of CARM1 alters the developmental programming of Glioma stem-like cells and creates a druggable NGFR/NTRK dependency

Dejauwne L. Young, Jennifer T. Aguilan, Ronald R. Cutler, Stephanie Stransky, Joseph D. DeAngelo, Jacob S. Roth, Beata Małachowska, Justin Vercellino, Brett I. Bell, David Shechter, Philip J. Tofilon, Richard E. Phillips, Chandan Guha, Simone Sidoli

This study shows that CARM1 loss in GSCs disrupts developmental programming, shifts lineage towards radial glia, and creates a dependency on NGFR/NTRK signaling, making them vulnerable to NTRK inhibitors.

bioRxiv · Apr 11, 2025 · PreprintCancer Biology