Author

Brett I. Bell

Albert Einstein College of MedicineORCID 0000-0002-8439-1402Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Cancer Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Organism:human· 1 Assay:proteomics· 1

Frequent Co-Authors

Beata MałachowskaiD· 1 shared Chandan GuhaiD· 1 shared David ShechteriD· 1 shared Dejauwne L. YoungiD· 1 shared Jacob S. RothiD· 1 shared Jennifer T. AguilaniD· 1 shared Joseph D. DeAngeloiD· 1 shared Justin VercellinoiD· 1 shared

Activity by Year

2025-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperCancer Biology

Beata MałachowskaORCID

Mutual co-authors in cluster

Author profile →

1 paper

Recent·Most cited

Loss of CARM1 alters the developmental programming of Glioma stem-like cells and creates a druggable NGFR/NTRK dependency

Dejauwne L. Young, Jennifer T. Aguilan, Ronald R. Cutler, Stephanie Stransky, Joseph D. DeAngelo, Jacob S. Roth, Beata Małachowska, Justin Vercellino, Brett I. Bell, David Shechter, Philip J. Tofilon, Richard E. Phillips, Chandan Guha, Simone Sidoli

This study shows that CARM1 loss in GSCs disrupts developmental programming, shifts lineage towards radial glia, and creates a dependency on NGFR/NTRK signaling, making them vulnerable to NTRK inhibitors.

bioRxiv · Apr 11, 2025 · PreprintCancer Biology