Author

Markus Christmann

Johannes Gutenberg University MainzORCID 0000-0002-9672-231XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Approach:experimental assay· 1 Approach:statistical model· 1

Frequent Co-Authors

Al‐Hassan M. MustafaiD· 1 shared Daniela PfeifferiD· 1 shared Georg Nagel· 1 shared Günter SchneideriD· 1 shared Huong Becker· 1 shared Jörg HartkampiD· 1 shared Kaveri Raja· 1 shared Magdalena C. LiebliD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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1 shared paperCancer Biology

Al‐Hassan M. MustafaORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

HIPK2 regulates homology-directed DNA repair and PARP inhibitor sensitivity

Patrick Weyerhäuser, Pierre‐Olivier Frappart, Georg Nagel, Stephan Krieg, Teodora Nikolova, Wynand Paul Roos, Kaveri Raja, Sahar Karbassi, Steffen Dickopf, Magdalena C. Liebl, Daniela Pfeiffer, Huong Becker, Yang He, Markus Christmann, Matthias Altmeyer, Thomas G. Hofmann

This study identifies HIPK2's role in HR repair and PARPi sensitivity by regulating BRCA1 stability, suggesting HIPK2 inhibition as a therapeutic strategy for BRCA1-proficient cancers.

bioRxiv · Jun 1, 2026 · PreprintCell Biology

The Deacetylases HDAC1 and HDAC2 Safeguard BCR-ABL-positive Cells from Replication Stress-Induced Apoptosis via the Nuclear to Mitochondrial p73-NOXA Axis

Al‐Hassan M. Mustafa, Miriam Pons, Nisintha Mahendrarajah, Maren Wiegerling, Jörg Hartkamp, Markus Christmann, Markus Philipp Radsak, Günter Schneider, Matthias Wirth, Oliver Holger Krämer

This study shows HDAC1 and HDAC2 protect BCR-ABL-positive CML cells from replication stress-induced apoptosis via the p73-NOXA axis.

bioRxiv · Dec 16, 2025 · PreprintCancer Biology