Author

Rebecca L. Murdaugh

Baylor College of MedicineORCID 0000-0002-9950-8634Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:chromatin & epigenetic regulation· 2 Organism:human· 2 Approach:statistical model· 2 Research Focus:cancer & tumor microenvironment· 1 Assay:ChIP-seq· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1

Frequent Co-Authors

Jamie N. AnastasiD· 3 shared Rosemary U. Richard· 3 shared Adam Fiseha Kebede· 2 shared Akdes Serin HarmancıiD· 2 shared Alan L. JiaoiD· 2 shared Barry M. ZeeiD· 2 shared Brittany R EberliD· 2 shared Caitlin L. BagnettoiD· 2 shared

Activity by Year

2026-2026 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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3 shared papersCancer Biology

Jamie N. AnastasORCID

Mutual co-authors in cluster

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3 papers

Recent·Most cited

Targeting SAGA/ATAC complexes disrupts chromatin regulation and cholesterol metabolism in diffuse midline glioma

Rosemary U. Richard, Caitlin L. Bagnetto, Rebecca L. Murdaugh, Brittany R Eberl, Alan L. Jiao, Adam Fiseha Kebede, Ena I. Campos-Hensley, Barry M. Zee, Margaret M. Harrington, Mariella G. Filbin, Akdes Serin Harmancı, Yang Shi, Jamie N. Anastas

This study shows that targeting SAGA/ATAC complexes disrupts chromatin regulation and cholesterol metabolism in diffuse midline glioma, leading to reduced tumor growth.

bioRxiv · Aug 17, 2026 · PreprintCancer Biology

Elongin B orchestrates chromatin and transcriptional programs in H3K27M-mutant diffuse midline glioma

Alan L. Jiao, Rebecca L. Murdaugh, Adam Fiseha Kebede, Claudia A. Mimoso, Barry M. Zee, Rosemary U. Richard, Kwanha Yu, Caitlin L. Bagnetto, Karen Adelman, Mariella G. Filbin, Yang Shi, Jamie N. Anastas

This study identifies elongin B as a key regulator of Pol2 elongation in H3K27M-mutant diffuse midline glioma, showing its role in maintaining chromatin and transcriptional dysregulation.

bioRxiv · May 19, 2026 · PreprintCancer Biology

H3 dopaminylation and CaMKII modulate diffuse midline glioma response to CDK9 inhibition

Rebecca L. Murdaugh, Brittany R Eberl, Rosemary U. Richard, Ena I. Campos-Hensley, Adanze N Nnyagu, W. Austin Elam, Ai-Ni Tsao, Jack R Tremblay, Ruiying Ma, Alfred K. Dei-Ampeh, Kieu Pham, Daniel C. Kraushaar, Kwanha Yu, Calla M. Olson, Akdes Serin Harmancı, , Jamie N. Anastas

This study shows that combining CDK9 inhibitors with neuropsychiatric drugs reduces diffuse midline glioma growth by altering H3 dopaminylation and CaMKII signaling.

bioRxiv · Apr 10, 2026 · PreprintCancer Biology