Franz G. Zingl

This paper presents two new antibody-recruiting molecule (ARM) strategies to combat multidrug-resistant bacterial infections using synthetic peptides and dimeric antibiotics.

The paper constructs a urinary microbiome catalog and identifies antimicrobial peptides from it that inhibit uropathogenic E. coli.

This study investigates how serotype differences in Vibrio cholerae O1 affect pathogenicity, finding that O1 antigen methylation enhances colonization and resistance to antimicrobial peptides.

The paper introduces MultiCAST, a high-throughput CRISPR-associated transposon system for bacterial gene disruption, enabling rapid and scalable genetic screens using amplicon sequencing.

The paper shows that anoxia activates the CRISPR-Cas system in the mouse intestine via the Fnr regulator, suggesting an adaptation for microbial defense.

This study shows that reduced motility in Vibrio cholerae decreases inter-animal transmission, while increased motility enhances transmission efficiency in mice.

This study identifies the Mla pathway as critical for Vibrio cholerae re-expansion from stationary phase by maintaining energy homeostasis and preventing outer membrane vesicle shedding.