Margarita Anisimova

The paper induces spike-timing-dependent plasticity in hippocampal synapses using optogenetics and performs ultrastructural analysis with transmission electron microscopy.

This study shows that D-serine inhibits non-ionotropic NMDA receptor signaling and plasticity by blocking glutamate-induced conformational changes in the absence of co-agonist.

This study shows that structural, not enzymatic, functions of CaMKII are required for activity-dependent stabilization of new dendritic spines.

The paper investigates the relationship between neuronal spiking frequency and FOS expression in hippocampal neurons, finding that FOS expression is cell-type specific, frequency-dependent, and not cell-autonomous.

The paper shows that reduced D-serine in a schizophrenia mouse model enhances non-ionotropic NMDAR signaling, leading to dendritic spine destabilization.

The paper uses optogenetic stimulation to induce spike-timing-dependent plasticity (STDP) and shows that late potentiation occurs regardless of spiking order, but requires ongoing activity.