Tobias Dörr

This paper introduces a digital droplet microarray system for high-throughput antibiotic tolerance measurement using SPOTs and Poissonian statistics, demonstrating its application to Klebsiella pneumoniae.

This study develops a mathematical model showing that bacterial corpses can influence infection dynamics and immune evolution by both hindering pathogen control and protecting against autoimmune damage.

This study identifies differential roles of ShyA and ShyC endopeptidases in Vibrio cholerae adaptation to low osmolarity and antibiotic stress.

This study identifies widespread meropenem tolerance in clinical Klebsiella pneumoniae isolates and reveals genetic mechanisms involving cell envelope stress responses and lytic transglycosylase MltB.

This paper identifies GlmU as a critical enzyme inhibited by sugar phosphates in Vibrio cholerae, linking glucose metabolism to cell envelope integrity and antibiotic susceptibility.

This study identifies three genetic factors in Enterobacter cloacae that collectively contribute to resistance against the antimicrobial peptide cecropin B, showing inoculum-dependent survival heterogeneity.

The paper measures the Young's modulus of bacterial cell envelopes in E. coli, V. cholerae, and S. aureus using a microfluidic system and inverse finite element analysis, finding values ranging from 0.12 to 2.06 MPa.

The paper uses computational models to show that host-pathogen feedbacks can lead to divergent infection outcomes, even with similar initial conditions.