Frédéric Barras

This study identifies a molecular link between iron-sulfur cluster biogenesis and fatty acid synthesis in E. coli through the interaction of ACP and IscS.

This study shows that mosquito larvae rely on gut microbiota for energy production, nitrogen excretion, and sterol metabolism, with bacterial vitamins and fatty acid degradation playing critical roles in larval development.

The paper introduces an automated framework for optimizing bacterial tracking pipelines using TrackMate 8, demonstrating its effectiveness on two use cases.

This study shows fumarate-based drugs modify gut bacteria via cysteine succination, affecting their functions and interactions in both isolated cultures and microbial communities.

This paper shows that 2,4-dienoyl-CoA reductase (FadH) is essential for E. coli to degrade unsaturated fatty acids in complex mixtures and that eukaryotic DECR enzymes can functionally replace FadH.

The paper identifies additional tRNA targets of the AriB nuclease in the PARIS system and shows that phage-encoded tRNAs can inhibit this defense.

This study shows that LdcI filamentation in E. coli drives peripheral clustering, enhancing acid stress response by spatially organizing the enzyme.

The paper shows that B vitamin supplementation during mosquito larval development affects adult sex ratio, with female enrichment, and identifies sex-specific toxicity and nutritional requirements.

The paper develops a logical model of Fe-S cluster biogenesis in E. coli to analyze how environmental stressors like iron deprivation and oxidative stress regulate this process.

The paper investigates why iron-sulphur cluster enzymes are often inactive when heterologously expressed in E. coli and identifies strategies to improve their functionality.

The paper shows RyhB, a small RNA, induces gentamicin resistance in E. coli under iron starvation by repressing respiratory complexes Nuo and Sdh.