Author

Shashi Ranjan

New York University Abu DhabiORCID 0000-0003-3667-1861Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 1 Developmental Biology· 1

Signature Assays, Organisms & Methods

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Assay:DNA methylation· 2 Approach:experimental assay· 2 Organism:zebrafish· 2 Research Focus:cancer & tumor microenvironment· 1

Frequent Co-Authors

Bhavani P. MadakashiraiD· 2 shared Elena MagnaniiD· 2 shared Kirsten C. SadleriD· 2 shared Charlene CheniD· 1 shared Filippo MacchiiD· 1 shared Sema Elif EskiiD· 1 shared Sumeet Pal SinghiD· 1 shared Tijana RandiciD· 1 shared

Activity by Year

2024-2024 · peak 2/yr

Co-Author Collaboration Network

8 top collaborators · 28 mutual co-author links

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2 shared papersCancer Biology

Bhavani P. MadakashiraORCID

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2 papers

Recent·Most cited

Cancer BiologybioRxivPreprintDec 29, 2024

Epigenetic Disordering Drives Stemness, Senescence Escape and Tumor Heterogeneity

Elena Magnani, Filippo Macchi, Tijana Randic…·Kirsten C. Sadler

The paper shows that high UHRF1 levels in zebrafish hepatocytes cause epigenetic disordering, leading to stemness, senescence escape, and tumor heterogeneity in hepatocellular carcinomas.

2 citations

zebrafishDNA methylation

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Developmental BiologyNucleic Acids ResearchPeer-reviewedFeb 7, 2024

DNA hypomethylation activates Cdk4/6 and ATR to cause dormant origin firing and cell cycle arrest that restricts liver outgrowth in zebrafish

Bhavani P. Madakashira, Elena Magnani, Shashi Ranjan, Kirsten C. Sadler

The paper shows DNA hypomethylation in zebrafish leads to Cdk4/6 activation, ATR-mediated replication stress, and cell cycle arrest, which restricts liver growth.

13 citations

zebrafishDNA methylation

𝕏 PostBluesky