Kätlin Silm

This study compares synaptic vesicle protein composition between dopamine and glutamate release sites, identifying differences in abundance and function that affect recycling kinetics.

This study identifies SONAVP neuron hyperactivity as a driver of aging-related homeostatic deficits and suggests AVP signaling modulation as a therapeutic strategy.

This study identifies AP-3 and VPS41 as factors that promote high-frequency dopamine release by targeting VMAT2 to axons and producing specialized synaptic vesicles.

The paper identifies a mechanism by which VGLUT1 increases synaptic vesicle clustering, reducing superpool size and spontaneous release frequency through interactions with endophilinA1 and intersectin1.