Author

Yuting Fu

University of South FloridaORCID 0009-0002-5833-2676Unclaimed ORCID profile · follow to get notified of new preprints

Additional preprints under this name without ORCID

Yuting Fu3 papers · Westlake University

Biology Disciplines

Click to filter author papers

Developmental Biology· 1 Pathology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Assay:multiome· 1 Assay:proteomics· 1

Frequent Co-Authors

George E. DavisiD· 2 shared Abbigail PriceiD· 1 shared Austin Mills· 1 shared Diandra M. MastrogiacomoiD· 1 shared Joshua Paul ScallaniD· 1 shared Kunyu LiiD· 1 shared Luz A. Knauer· 1 shared Michael T. DellingeriD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersPathology

George E. DavisORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

PathologyArteriosclerosis Thrombosis and Vascular BiologyPeer-reviewedMar 26, 2026

Lymphatic Malformations with Activating KRAS Mutations Impair Lymphatic Valve Development Through Matrix Metalloproteinases

Diandra M. Mastrogiacomo, Abbigail Price, Yuting Fu…·Joshua Paul Scallan

This study shows that KRAS mutations impair lymphatic valve development by upregulating MMPs, which degrade the valve ECM core.

DOI 10.1161/atvbaha.126.324257

mouseproteomics

𝕏 PostBluesky

Developmental BiologyArteriosclerosis Thrombosis and Vascular BiologyPeer-reviewedJun 12, 2025

Lats1/2 are essential for developmental vascular remodeling and biomechanical adaptation to shear stress

Mitzy A. Cowdin, Tuli Pramanik, Shelby R. Mohr-Allen…·Ondine B. Cleaver

This paper shows that Lats1/2 are essential for vascular remodeling and response to shear stress in embryonic endothelial cells, leading to vascular defects and embryonic lethality.

9 citationstop 4%

mouseexperimental assay

𝕏 PostBluesky