Author

Hana Bader

University of PennsylvaniaORCID 0009-0006-4289-5836Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 2 Approach:experimental assay· 2 Organism:human· 2 Assay:proteomics· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1

Frequent Co-Authors

Lukasz J. BugajiD· 2 shared Alexander Byrd· 1 shared Anh Le· 1 shared David Gonzalez-MartineziD· 1 shared Nidhi SahniiD· 1 shared Robert Charles DoebeleiD· 1 shared Sofia Wissert· 1 shared Yueying Gu· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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2 shared papersBioengineering

Lukasz J. BugajORCID

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2 papers

Recent·Most cited

BioengineeringACS Synthetic BiologyPeer-reviewedJan 30, 2026

Optogenetic translocation to subcellular compartments through regulation of protein avidity

Zikang Dennis Huang, Yueying Gu, Yuzhi Carol Gao…·Lukasz J. Bugaj

This paper introduces Aviatar, a single-component optogenetic system for inducible protein translocation to various subcellular compartments, demonstrating its utility in regulating actin polymerization and cancer-related signaling.

1 citation

humanproteomics

𝕏 PostBluesky

Cancer BiologybioRxivPreprintNov 19, 2025

EGFR suppression and drug-induced potentiation are widespread features of oncogenic RTK fusions

Yuzhi Gao, David Gonzalez-Martinez, Sofia Wissert…·Lukasz J. Bugaj

The paper shows that RTK fusions suppress EGFR and sequester Grb2, and targeted therapies reverse this suppression, enhancing EGFR signaling and drug response.

3 citations

humanproteomics

𝕏 PostBluesky