Author

Steffen Joachim Sonnentag

Karlsruhe Institute of TechnologyORCID 0009-0008-4930-1150Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 2 Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Assay:organoid / primary culture· 1

Frequent Co-Authors

Dominique Hoch· 2 shared Geoffroy AndrieuxiD· 2 shared Véronique Orian‐RousseauiD· 2 shared Yvonne M. HenekaiD· 2 shared Alessandra GarganoiD· 1 shared Dirk GrimmiD· 1 shared Eliana StanganelloiD· 1 shared Florian R. GreteniD· 1 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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2 shared papersCancer Biology

Dominique Hoch

Mutual co-authors in cluster

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2 papers

Recent·Most cited

Cancer BiologyCell Death and DiseasePeer-reviewedAug 15, 2026

Loss of CD44 re-educates pancreatic cancer-associated fibroblasts modulating their fibrotic and immunosuppressive functions

Sven Máté Treffert, Yvonne M. Heneka, Julie Martin…·Véronique Orian‐Rousseau

This study shows that CD44 deletion in pancreatic cancer-associated fibroblasts reduces tumor volume, alters their morphology and function, and enhances anti-tumor immune responses.

DOI 10.1038/s41419-026-09155-5

mousehuman

𝕏 PostBluesky

Cancer BiologybioRxivPreprintSep 27, 2024

Breaking the cycle: How targeting CD44v6/MET signaling disrupts colorectal cancer cell plasticity

Steffen Joachim Sonnentag, Selina Goldbach, Dominique Hoch…·Véronique Orian‐Rousseau

This study shows that targeting CD44v6/MET signaling disrupts colorectal cancer cell plasticity by repressing Lgr5+ cell reappearance and affecting YAP/TAZ signaling.

DOI 10.1101/2024.09.27.614761

mouseorganoid / primary culture

𝕏 PostBluesky