Author

Ying C. Li

University of California, San FranciscoORCID 0009-0009-4792-8336Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Research Focus:drug response / drug discovery· 2 Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Assay:scRNA-seq· 1 Approach:statistical model· 1

Frequent Co-Authors

Kevin J. BenderiD· 2 shared Andrew D. NelsoniD· 1 shared Atehsa SahaguniD· 1 shared Beniamin KrupkiniD· 1 shared Caroline M. Keeshen· 1 shared Christina K. KimiD· 1 shared Coline ArnouldiD· 1 shared Congyi LuiD· 1 shared

Activity by Year

2025-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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2 shared papersNeuroscience

Kevin J. BenderORCID

Mutual co-authors in cluster

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2 papers

Recent·Most cited

CRISPR activation rescues abnormalities in SCN2A haploinsufficiency-associated autism spectrum disorder

Serena Tamura, Andrew D. Nelson, Perry W.E. Spratt, Elizabeth C. Hamada, Xujia Zhou, Henry Kyoung, Zizheng Li, Coline Arnould, Vladyslav Barskyi, Beniamin Krupkin, Kiana Young, Jingjing Zhao, Stephanie S. Holden, Atehsa Sahagun, Caroline M. Keeshen, , Kevin J. Bender

This study shows that CRISPR activation can rescue electrophysiological deficits in SCN2A haploinsufficiency by increasing the expression of the remaining functional allele in mice and human neurons.

Nature · Sep 17, 2025Peer-reviewedNeuroscience

D3 dopamine receptors implicate a striatal neuronal subtype in the aversive effects of the antipsychotic drug quetiapine

Elinor Lewis, Jessie Muir, Ying C. Li, Madeline King, Kaden P. Adams, Julianna Glienke, Sarah Warren Gooding, Kevin J. Bender, Christina K. Kim, Jennifer Lynne Whistler

This study identifies D3R-expressing neurons in the NAc as mediators of quetiapine's aversive effects, showing that chronic treatment reduces this aversion.

bioRxiv · Apr 15, 2025 · PreprintNeuroscience