Author

X. X. Ding

University of California, DavisORCID 0009-0009-4831-1274Unclaimed ORCID profile · follow to get notified of new preprints

Additional preprints under this name without ORCID

Xiangyi Ding1 paper · University of California, Davis

Biology Disciplines

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Cell Biology· 2

Signature Assays, Organisms & Methods

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Organism:C. elegans· 2 Research Focus:cell type annotation· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:development & morphogenesis· 1 Approach:experimental assay· 1 Approach:genetic perturbation / CRISPR· 1 Assay:imaging· 1

Frequent Co-Authors

Daniel A. StarriD· 2 shared G. W. Gant LuxtoniD· 2 shared Daniel ElnataniD· 1 shared Ellen F. GregoryiD· 1 shared Guanzhi Wang· 1 shared Matthew Laurence· 1 shared Megan Couture· 1 shared S Krishna KumariiD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersCell Biology

Daniel A. StarrORCID

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2 papers

Recent·Most cited

Cell BiologybioRxivPreprintJul 22, 2026

Nuclear envelope-dependent heterochromatin positioning gates the timing of nucleolar assembly in the early embryo

X. X. Ding, Guanzhi Wang, Daniel Elnatan…·G. W. Gant Luxton

The paper shows that the LINC complex and nuclear lamina delay nucleolar assembly in C. elegans embryos by organizing heterochromatin, with CEC-4 playing a key role in suppressing premature nucleoli.

DOI 10.64898/2026.07.20.739428

C. elegansgenetic perturbation / CRISPR

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Cell BiologybioRxivPreprintSep 22, 2025

Muscular dystrophy-associated lamin variants disrupt cellular organization through a nucleolar-ribosomal axis controlling cytoplasmic macromolecular crowding

Xiangyi Ding, S Krishna Kumari, Ellen F. Gregory…·G. W. Gant Luxton

This study identifies a nucleolar-ribosomal axis linking lamin mutations to cytoplasmic biophysical changes in EDMD, revealing a new pathway for laminopathy pathogenesis.

3 citations

C. elegansimaging

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