Author

A. M. Balynas

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Assay:electrophysiology· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1

Frequent Co-Authors

D. Chaudhuri· 2 shared X. Yin· 2 shared A. C. Corbin· 1 shared C. H. Selzman· 1 shared C. P. Kyriakopoulos· 1 shared D. R. Eberhardt· 1 shared E. Balderas· 1 shared E. Rekate· 1 shared

Activity by Year

2021-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

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2 shared papersCell Biology

D. Chaudhuri

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2 papers

Recent·Most cited

Mitochondria possess a large, non-selective ionic current that is enhanced during cardiac injury.

E. Balderas, S. H. J. Lee, T. S. Shankar, X. Yin, A. M. Balynas, C. P. Kyriakopoulos, C. H. Selzman, S. G. Drakos, D. Chaudhuri

The paper identifies a large, non-selective ionic current in mitochondria, called LUNA, which increases during cardiac injury and is inhibited by amiloride.

bioRxiv · Nov 15, 2023 · PreprintCell Biology

EFHD1 ablation reduces cardiac mitoflash activation and protects cardiomyocytes from ischemia.

D. R. Eberhardt, S. H. Lee, X. Yin, A. M. Balynas, E. Rekate, J. N. Kraiss, M. J. Lang, M. A. Walsh, M. E. Streiff, A. C. Corbin, K. Funai, F. B. Sachse, D. Chaudhuri

This study shows that EFHD1 deletion reduces mitochondrial Ca2+ overload and ROS production, protecting cardiomyocytes from ischemia-induced injury.

bioRxiv · Oct 18, 2021Cell Biology