Author

A. W. Caulk

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Bioengineering· 2

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Approach:statistical model· 1

Frequent Co-Authors

J. D. Humphrey· 2 shared M. Latorre· 2 shared S.-I. Murtada· 2 shared A. B. Ramachandra· 1 shared A. Rojas· 1 shared B. Jiang· 1 shared B. Spronck· 1 shared C.-S. He· 1 shared

Activity by Year

2019-2020 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 35 mutual co-author links

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2 shared papersBioengineering

J. D. Humphrey

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2 papers

Recent·Most cited

Genetic Background Dictates Aortic Fibrosis in Hypertensive Mice

B. Spronck, M. Latorre, S. Mehta, A. W. Caulk, A. B. Ramachandra, S.-I. Murtada, A. Rojas, C.-S. He, B. Jiang, M. Wang, M. R. Bersi, G. Tellides, J. D. Humphrey

This study shows that genetic background strain influences aortic fibrosis in hypertensive mice, with C57BL/6J mice showing more maladaptive remodeling than 129SvEv mice.

bioRxiv · May 5, 2020Bioengineering

Paradoxical Aortic Stiffening and Subsequent Cardiac Dysfunction in Hutchinson-Gilford Progeria Syndrome

S.-I. Murtada, Y. Kawamura, A. W. Caulk, H. Ahmadzadeh, N. Mikush, K. Zimmerman, D. Kavanagh, D. Weiss, M. Latorre, Z. W. Zhuang, G. S. Shadel, D. T. Braddock, J. D. Humphrey

This study shows that aortic stiffening and cardiac dysfunction in HGPS mice occur due to loss of smooth muscle function and proteoglycan accumulation, independent of sex, with potential therapeutic implications.

bioRxiv · Oct 2, 2019Bioengineering