Author

K. Zimmerman

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Bioengineering· 1 Cell Biology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:cell signaling & trafficking· 2 Research Focus:development & morphogenesis· 2 Research Focus:host-pathogen & immune response· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1

Frequent Co-Authors

A. Pringle· 1 shared A. W. Caulk· 1 shared C. A. Adams· 1 shared C. Allamargot· 1 shared D. Abhinav· 1 shared D. Kavanagh· 1 shared D. T. Braddock· 1 shared D. Weiss· 1 shared

Activity by Year

2019-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 13 mutual co-author links

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1 shared paperMicrobiology

A. Pringle

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Cardiac Specific Overexpression of Transcription Factor EB (TFEB) in Normal Hearts Induces Pathologic Cardiac Hypertrophy and Lethal Cardiomyopathy

H. C. Kenny, E. T. Weatherford, G. V. Collins, C. Allamargot, T. Gesalla, K. Zimmerman, H. Goel, J. M. McLendon, D.-F. Dai, T. Romac, J. Streeter, D. Abhinav, R. O. Pereira, J. Sharafuddin, L.-S. Song, E. D. Abel

This study shows that cardiac-specific overexpression of TFEB leads to pathologic hypertrophy and lethal cardiomyopathy through mitochondrial dysfunction and fibrosis.

bioRxiv · Feb 16, 2021 · PreprintCell Biology

Fungal seed pathogens of wild chili peppers possess multiple mechanisms to tolerate capsaicinoids

C. A. Adams, K. Zimmerman, K. Fenstermacher, M. G. Thompson, W. Skyrud, S. W. Behie, A. Pringle

This study investigates how fungal seed pathogens of wild chili peppers tolerate capsaicinoids, finding alternative respiratory enzymes and membrane disruption as potential mechanisms.

Applied and Environmental Microbiology · Jan 21, 2020Peer-reviewedMicrobiology

Paradoxical Aortic Stiffening and Subsequent Cardiac Dysfunction in Hutchinson-Gilford Progeria Syndrome

S.-I. Murtada, Y. Kawamura, A. W. Caulk, H. Ahmadzadeh, N. Mikush, K. Zimmerman, D. Kavanagh, D. Weiss, M. Latorre, Z. W. Zhuang, G. S. Shadel, D. T. Braddock, J. D. Humphrey

This study shows that aortic stiffening and cardiac dysfunction in HGPS mice occur due to loss of smooth muscle function and proteoglycan accumulation, independent of sex, with potential therapeutic implications.

bioRxiv · Oct 2, 2019Bioengineering