Author

C. G. Weindel

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Approach:experimental assay· 4 Organism:mouse· 4 Research Focus:host-pathogen & immune response· 3 Assay:bulk RNA-seq· 2 Research Focus:cell signaling & trafficking· 2 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Research Focus:development & morphogenesis· 1

Frequent Co-Authors

K. L. PatrickiD· 4 shared R. O. WatsoniD· 4 shared K. J. Vail· 2 shared S. Azam· 2 shared S. L. Bell· 2 shared A. Coleman· 1 shared A. Devigne· 1 shared A. K. Coleman· 1 shared

Activity by Year

2019-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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4 shared papersImmunology

K. L. PatrickORCID

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4 papers

Recent·Most cited

The RNA binding protein, HNRNPA2B1, regulates IFNG signaling in macrophages

M. Mohammed Salih, C. G. Weindel, E. Malekos, L. Sudek, S. Katzman, C. J. Mabry, A. Coleman, S. Azam, R. O. Watson, K. L. Patrick, S. Carpenter

This study shows HNRNPA2B1 regulates IFNG signaling in macrophages by controlling IFNG receptor splicing and bacterial restriction.

bioRxiv · Oct 12, 2023 · PreprintImmunology

The early macrophage response to pathogens requires dynamic regulation of the nuclear paraspeckle

S. Azam, K. S. Armijo, C. G. Weindel, A. Devigne, S. Nakagawa, T. Hirose, S. L. Carpenter, R. O. Watson, K. L. Patrick

This paper shows that the nuclear paraspeckle dynamically regulates inflammatory gene expression in macrophages, essential for controlling pathogen replication.

bioRxiv · May 11, 2023Immunology

Mitochondrial dysfunction promotes alternative gasdermin D-mediated inflammatory cell death and susceptibility to infection

C. G. Weindel, X. Zhao, E. Martinez, S. L. Bell, K. J. Vail, A. K. Coleman, J. J. VanPortfliet, B. Zhao, C. Mabry, P. Li, P. West, J. Karpac, K. L. Patrick, R. O. Watson

This study shows that mitochondrial dysfunction caused by Lrrk2G2019S promotes non-pyroptotic cell death via GSDMD and increases susceptibility to infection.

bioRxiv · Nov 18, 2021 · PreprintImmunology

LRRK2 regulates innate immune responses and neuroinflammation during Mycobacterium tuberculosis infection

C. G. Weindel, S. L. Bell, T. E. Huntington, K. J. Vail, R. Srinivasan, K. L. Patrick, R. O. Watson

This study shows LRRK2 regulates immune responses and neuroinflammation during Mycobacterium tuberculosis infection, linking it to neurodegenerative disease mechanisms.

bioRxiv · Jul 26, 2019Immunology