Author

C. Montagna

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 4 Cell Biology· 2

Signature Assays, Organisms & Methods

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Organism:human· 5 Approach:experimental assay· 4 Assay:bulk RNA-seq· 3 Assay:proteomics· 3 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:statistical model· 2 Research Focus:cancer & tumor microenvironment· 1

Frequent Co-Authors

E. T. Spiliotis· 2 shared A. Lischka· 1 shared A. T. Madsen· 1 shared A. Van Arsdale· 1 shared A. Verma· 1 shared B. Harmon· 1 shared B. M. Ghadimi· 1 shared D. Angelis· 1 shared

Activity by Year

2019-2023 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 8 mutual co-author links

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2 shared papersCell Biology

E. T. Spiliotis

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Author profile →

6 papers

Recent·Most cited

An oncogenic isoform of septin 9 promotes the formation of juxtanuclear invadopodia by reducing nuclear deformability

J. Okletey, D. Angelis, T. M. Jones, C. Montagna, E. T. Spiliotis

This paper shows that the oncogenic SEPT9_i1 isoform promotes juxtanuclear invadopodia formation by reducing nuclear deformability in breast cancer cells.

bioRxiv · Jun 18, 2023Cell Biology

Functional Contribution of Cancer-Associated Fibroblasts in Glioblastoma

P. M. Galbo, Y. Liu, M. Peng, Y. Wei, A. T. Madsen, S. Graff, C. Montagna, J. E. Segall, S. Sidoli, X. Zang, D. Zheng

This study identifies and characterizes cancer-associated fibroblasts (CAFs) in glioblastoma using single-cell RNA sequencing and spatial transcriptomics, revealing their functional roles in tumor progression and microenvironment interactions.

bioRxiv · Apr 7, 2022 · PreprintCancer Biology

Deletion of PI3-Kinase Promotes Myelodysplasia Through Dysregulation of Autophagy in Hematopoietic Stem Cells

K. Ames, I. Kaur, Y. Shi, M. M. Tong, T. Sinclair, S. Hemmati, S. G. Glushakow-Smith, E. E. Tein, L. Gurska, R. Dubin, J. Shan, K. Pradhan, A. Verma, C. Montagna, K. Gritsman

This study shows that deleting all three Class IA PI3K isoforms in HSCs leads to myelodysplasia through impaired autophagy, which affects differentiation and multilineage repopulation.

bioRxiv · Dec 4, 2020 · PreprintCancer Biology

Insertional oncogenesis by HPV70 revealed by multiple genomic analyses in a clinically HPV-negative cervical cancer

A. Van Arsdale, N. E. Patterson, E. C. Maggi, L. Agoni, K. Van Doorslaer, B. Harmon, N. Nevadunsky, D. Y. S. Kuo, M. H. Einstein, J. Lenz, C. Montagna

The paper identifies HPV70 insertion into BCL11B in an HPV-negative cervical cancer through genomic analyses, suggesting HPV70's oncogenic potential and novel BCL11B variants.

bioRxiv · May 10, 2019Cancer Biology

Proteomic profiling of the oncogenic septin 9 reveals isoform-specific interactions in breast cancer cells

L. Devlin, G. Perkins, J. R. Bowen, C. Montagna, E. T. Spiliotis

The paper identifies isoform-specific interactions of SEPT9 in breast cancer cells through proteomic analysis, revealing distinct molecular networks and functions for different SEPT9 variants.

bioRxiv · Mar 5, 2019Cell Biology

Cancer-type specific aneuploidies hard-wire chromosome-wide gene expression patterns of their tissue of origin

N. Auslander, K. Heselmeyer-Haddad, S. Patkar, D. Hirsch, J. Camps, M. Brown, D. Bronder, W.-D. Chen, R. Lokanga, D. Wangsa, D. Wangsa, Y. Hu, A. Lischka, R. Braun, G. Emons, , t. ried

The paper shows cancer-type specific aneuploidies correlate with normal tissue gene expression patterns, suggesting they hard-wire chromosome-wide expression levels to support tumorigenesis.

bioRxiv · Mar 1, 2019Cancer Biology