Author

Claudia Krenner

Ludwig-Maximilians-Universität München

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Organism:human· 1 Research Focus:protein structure prediction· 1

Frequent Co-Authors

Anika ReifschneideriD· 2 shared Anja CapelliD· 2 shared Christian HaassiD· 2 shared Gilbert Di PaoloiD· 2 shared Katrin Buschmann· 2 shared Alessio RicciiD· 1 shared Andrea Ahles· 1 shared Arthur LiesziD· 1 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 41 mutual co-author links

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2 shared papersNeuroscience

Anika ReifschneiderORCID

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2 papers

Recent·Most cited

TREM2 expression level is critical for microglial state, metabolic capacity and efficacy of TREM2 agonism

Astrid Feiten, Kilian Dahm, Kai Schlepckow, Bettina van Lengerich, Jung H. Suh, Anika Reifschneider, Benedikt Wefers, Laura M. Bartos, Karin Wind-Mark, Lis de Weerd, T Ulas, Elena De Domenico, Pia Grundschöttel, Stefan Paulusch, Benjamin Tast, , Christian Haass

This study investigates how TREM2 expression levels affect microglial function, metabolism, and response to TREM2 agonists in a mouse model of Alzheimer's disease.

Nature Communications · Jan 24, 2026Peer-reviewedNeuroscience

Enhanced legumain activity links progranulin deficiency to TDP-43 pathology in frontotemporal lobar degeneration

Sophie Robinson, Marvin Reich, Maria‐Teresa Mühlhofer, Katrin Buschmann, Eline Wauters, Quirin Mühlhofer, Georg Werner, Andrea Ahles, Stefan Hanns Engelhardt, Claudia Krenner, Björn Bartels, Simon Gutbier, Anika Reifschneider, Henrick Riemenschneider, Thomas Reinheckel, , Anja Capell

This study shows that reduced progranulin leads to increased legumain activity, which promotes TDP-43 pathology in FTLD, suggesting legumain as a therapeutic target.

bioRxiv · Jan 16, 2024 · PreprintNeuroscience