Author

Daphne Mayer

Sanford Burnham Prebys Medical Discovery Institute

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cancer Biology· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:development & morphogenesis· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1

Frequent Co-Authors

Alessandra CecchiniiD· 2 shared Alessandra SaccoiD· 2 shared Alexandre R. ColasiD· 2 shared Cedomir Stamenkovic· 2 shared Chiara NicolettiiD· 2 shared Gabriele Guarnaccia· 2 shared Luca CaputoiD· 2 shared Prem PuriiD· 2 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersCancer Biology

Alessandra CecchiniORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Tumor-derived SAA1-TLR4 signaling drives tumor-to-muscle communication in pancreatic cancer cachexia

Gabriele Guarnaccia, Jimmy Massenet, Alessandra Cecchini, Elena Guarnaccia, Beatrice Silvestri, Chiara Nicoletti, Jesus R Barajas, David Sala, Marcos G. Teneche, Brightany Li, Luca Caputo, Cedomir Stamenkovic, Tatiana M. Moreno, Rabi Murad, Shawn Delaware, , Alessandra Sacco

This study identifies SAA1-TLR4 signaling as a tumor-derived pathway driving muscle wasting in pancreatic cancer, showing that targeting this pathway can preserve muscle mass and improve survival without affecting tumor growth.

bioRxiv · Sep 9, 2026 · PreprintCancer Biology

Tenascin-C from the tissue microenvironment promotes muscle stem cell self-renewal through Annexin A2

Alessandra Cecchini, Mafalda Loreti, Collin D. Kaufman, Cedomir Stamenkovic, Gabriele Guarnaccia, Alma Renero, Chiara Nicoletti, Anaïs Kervadec, Daphne Mayer, Shawn Delaware, Luca Caputo, Xiuqing Wei, Alexandre R. Colas, Prem Puri, Alessandra Sacco

This study identifies Tenascin-C as a key factor promoting muscle stem cell self-renewal through Annexin A2 signaling, showing its role in muscle repair and aging.

Communications Biology · Dec 5, 2025Peer-reviewedCell Biology